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The hepatic extracellular matrix (ECM) consists of a diverse range of components that respond dynamically to injury to facilitate wound healing. However, perturbed or incomplete restitution of damage can lead to an altered ECM and collagenous scar formation (i.e., fibrosis).  It is our goal to better understand this process, as well as to develop minimally-invasive biomarkers to predict interindividual risk of severe liver disease.